IS IT DIZZINESS OR VERTIGO?
Vertigo is a type of dizziness felt like a false sensation of movement, like motion sickness or dizziness. The most common causes of vertigo are benign paroxysmal positional vertigo (BPPV), Meniere’s disease, and acute onset vertigo.
Symptoms of the vertigo-associated disease include nausea, vomiting, headache, stumbling while walking.
There are two categories of vertigo. Peripheral vertigo is the most common type and occurs due to a problem in the inner ear or the vestibular nerve. The vestibular nerve connects the inner ear with the brain. While sometimes a one-off episode, it is more commonly a generally sudden hallucination of motion over hours or days, often with nausea, headaches or double vision. The central form is due to a disease originating from the central nervous system (CNS), often including lesions of cranial nerve VIII. Vertigo is a type of dizziness that felt like a false sensation of movement.
Causes of peripheral vertigo:
Benign paroxysmal positional vertigo (BPPV), Meniere’s disease, is an inner ear disorder that affects balance and hearing. Acute peripheral vestibulopathy (APV) is inflammation of the inner ear, which causes a sudden onset of vertigo.
Rarely, peripheral vertigo is caused by: perilymphatic fistula, or abnormal communication between the middle ear and the inner ear, cholesteatoma erosion, or erosion caused by a cyst in the inner ear, otosclerosis, or abnormal bone growth in the middle ear.
Causes of central vertigo include stroke, tumour in the cerebellum, migraine, multiple sclerosis.
DIZZINESS AND VISION
Dizziness could also be a symptom of a serious brain problem such as stroke, bleeding in the brain or multiple sclerosis. Attacks are a fairly common symptom and side effect of multiple sclerosis (MS), occurring in about 20 per cent of people with MS at some point.
Dizziness is often confused or used interchangeably. Feeling dizzy involves unsteadiness, light-headedness, uneasiness or fatigue, whereas episodes of vertigo symptoms of spinning, the sensation of movement or whirling can occur when one is not actually moving.
Head movements and maneuvers such as Epley, Semont, Foster, and Brandt-Daroff techniques are often employed to diagnose and treat vertigo.
We use peripheral vision as in our driving vision, importantly automatically maintaining a stable spatial world to position ourselves to look around and see in the line of sight with central vision. The two visual systems work together, giving stable spatial structure and giving central vision detail. Eye muscle imbalances can add stress to the visual system.
A mismatch can occur between reality, and the signals your eyes, inner ears, and sense of touch send your brain.
The four classic symptoms are vertigo, tinnitus, a feeling of fullness or pressure in the ear, and fluctuating hearing or hearing loss.
If the vestibular nerve has been damaged by a viral infection causing conditions jointly known as vestibular neuronitis, vestibular neuritis or labyrinthitis, the result can be an infection of the vestibular nerve in the inner ear. As the vestibular nerve becomes inflamed, control and sense of balance can suffer, e.g. chronic dizziness. Vestibular blocking agents are sometimes used.
DIX HALLPIKE MANOEUVRE TO DIAGNOSE BPPV
The most common peripheral form is benign paroxysmal positional vertigo (BPPV) if no stroke or hypertension.
BPPV comes on quickly, causing disorientation and stumbling; some even fall out of bed. Most episodes last about a minute and recur over a period of a few days or weeks. The Dix-Hallpike manoeuvre can differentiate an inner ear problem specifically as different types of BPPV cause different eye movements and elicit symptoms of dizziness during a 45-degree head turn for at least 30 seconds.
The Head impulse is used as certain rapid eye movements can indicate a problem in the semicircular canals of the inner ear.
The Romberg test may show how vision will compensate for the loss of position sense as sway or fall can occur with eyes closed
Causations can be from simple optical illusions to complex brain tumours
Integration of vestibular system, somatosensory and visual input to maintain posture and balance is required. Visual, head and body movement training may be required.
Anything that disturbs this combination can disturb our spatial stability, especially reduced visual acuity, field loss, stroke, head injury,
The human ear is the key to dizziness. In the positional form, the eyes can provide clues.
A patient who has a problem with proprioception can still maintain balance by using vestibular function and vision.
In the Romberg test, the standing patient is asked to close his or her eyes.
An increased loss of balance is interpreted as a positive Romberg’s test suggestive of mild lesions of the sensory, vestibular, or proprioceptive systems. Migraine headaches can bring on the dizzying sensation, as can lie in an MRI machine’s magnetic field. Meniere’s disease is caused by a buildup of fluid in the inner ear. It can cause dizziness and ringing in the ears and hearing loss, sometimes alleviated by a low sodium diet and a diuretic to decrease fluid pressure in the inner ear.
Antiemetics such as meclizine may inhibit nausea or vomiting that accompanies most types of the condition.
The smoothness of pursuit eye movements, horizontally and vertically, and loss of fixation or saccadic intrusions are important as eye movements are often the first sign of these conditions and the most sensitive indicator of deterioration. The visual sense can compensate for this loss of proprioception of muscle and joint position.
Proprioception is the sense of the relative position of one’s own parts of the body and the strength of effort being employed in movement. Proprioceptors provide it in skeletal striated muscles and tendons and the fibrous membrane in joint capsules.
If caused by a dorsal column disorder, the patient may correct balance problems by opening his or her eyes.
If the lesion is in the cerebellum rather than the dorsal columns, the cerebellar ataxia of balance will not be corrected by visual compensation, as is the case in the sensory ataxia of the dorsal column.
Vertigo can be concurrent with paralysis.
Radiation treatment Pre and post-surgery
Vestibular migraine diagnosis can involve neurologists, OT and physiotherapy specialist clinics for balance and vestibular issues (visual-spatial movement affecting balance) and optometrists. Brain imaging, the trial of medications, and assessment for BPPV (benign paroxysmal positional vertigo, were standing up quickly from sitting or lying, or rolling over, can cause dizziness or vertigo), Epley manoeuvre treatments are typically done.
Visual function and ocular health can consider other primary or contributing factors. History of whiplash, concussion, head injury, pattern glare ( sensitivity to flickering light, striped shirts and carpet patterns, computer screens, Venetian blinds, lines of the small print move when reading. Visual motion sensitivity can be helped with chromatic filters, small degrees of prism in various ways, binasal sectoral occlusion, appropriate prescription, vision therapy.
BENIGN PAROXYMAL POSITIONAL VERTIGO (BPPV)
BPPV can occur when calcium builds up in canals of the inner ear, usually brought on by trauma to the head or by moving the head in certain positions.
The majority of peripheral vertigo is caused by otoconia (also called canals), tiny limestone and protein crystals that reside deep inside your ear in the vestibule dislodge from external motion to the gel in the utricle and migrate into one or more of the 3 fluid-filled semicircular canals instead.
The most commonly used and successful physical therapy for BPPV and treatment is the Epley manoeuvres (also known as a canalith repositioning procedure). The goal is to return the dislodged otoconia to the vestibule to lessen risk of falls and driving accidents utilising head positions designed to remove or shift inner ear ‘crystals’ in BPPV
Other treatments helping to control balance include calcium channel blockers, beta-blockers and tricyclic antidepressants.
Migraine prevention medication.
Medication to dampen the sensations of dizziness.
Vestibular compensation and vestibular rehabilitation
Training the brain to cope with the disorientating signals coming from the inner ears by learning to rely more on alternative signals coming from the eyes, ankles, legs and neck to maintain balance.
Diagnosis can be made by magnifying and measuring rapid eye movements with high definition recording googles during dizzy spells as crystals in the ear are moving in and out of place (e.g. nystagmus – and uncontrolled eye movement), the eye movements with physical therapy can be measured.
This may result to maintain orientation, e.g. during the Fukuda step test, which requires body movements with the eyes closed to see if the body strays from the midline were affected by the condition.
Sometimes combinations of problems such as sleep issues, anxiety, depression, PTSD, photophobia inside and out (which is referred to as pattern glare), motion sickness, visual motion sensitivity (VMS) can benefit by neuro-optometric rehabilitation assessment. Treatment might include spectacle prescription, a specifically determined tint, prisms in various ways, sectoral occlusion sometimes, and other options.
The aim is to improve function in activities of daily living (ADL’s) and quality of life (QOL).